We compute the four-point amplitude with external adjoint particles in N=2 SCQCD at two loops using N=1 superspace P03 Feynman diagrams, extending the results of arXiv:1406.7283.We consider the diagrammatic difference with the corresponding process of N=4 SYM finding a non-vanishing result, which is a non-trivial function of the kinematic variables
Vascular FLRT2 regulates venous-mediated angiogenic expansion and CNS barriergenesis
Abstract Veins have emerged as the origin of all Gum Stimulators other endothelial cell subtypes needed to expand vascular networks during developmental and pathological neoangiogenesis.Here, we uncover the role of the angioneurin Fibronectin Leucine Rich Transmembrane protein (FLRT) 2 in central nervous system (CNS) vascular development in the mou
Microcirculation disorders and structural and functional left ventricular abnormalities in patients with type 1 diabetes complicated by ketoacidosis
Aim.To assess the patterns of microcirculation disorders and its effect on the left ventricular (LV) structure and function in patients with type 1 diabetes (T1D) complicated by diabetic ketoacidosis (DKA).Material and methods.The study included 128 patients with T1D complicated by DKA.Echocardiography was performed according to the standard techni
Red blood cell-tumor cell interactions promote tumor cell progression
Abstract A critical step in the metastatic cascade is the survival of circulating tumor cells (CTCs) within the bloodstream.Although interactions between CTCs and various hematopoietic cells have been described, the role of red blood cells (RBCs) remains underexplored.This study investigated the interactions between tumor cells and RBCs from breast
Early developmental alterations of CA1 pyramidal cells in Dravet syndrome
Dravet Syndrome (DS) is most often caused by heterozygous loss-of-function mutations in the voltage-gated sodium channel gene SCN1A (Nav1.1), resulting in severe epilepsy and neurodevelopmental impairment thought to be cause by reduced interneuron excitability.However, recent studies in mouse models suggest that interneuron dysfunction alone does n